Magnetic Reconnection in Turbulent Plasmas and Gamma Ray Bursts

We discuss how the model of magnetic reconnection in the presence of turbulence proposed inLazarian & Vishniac 1999 makes the reconnection rate independent either of resistivity or microscopic plasma effects, but determined entirely by the magnetic field line wandering induced by turbulence. We explain that the model accounts for both fast and slow regimes of reconnection and that this property naturally induces flares of reconnection in low beta plasma environments. In addition, we show that the model involves volume reconnection which can convert a substantial part of the energy into energetic particles. It is important that the reconnection induces an efficient acceleration of the first order Fermi type. Finally, we relate the properties of the reconnection with the observed properties of gamma ray bursts and provide evidence supporting the explanation of gamma ray bursts based on energy release via reconnection.Comment: 12 pages, review talk at 5th International Symposium on High-Energy Gamma-Ray Astrophysics, Heidelber

Cosmic Ray transport in MHD turbulence: large and small scale interactions

Cosmic ray (CR) transport and acceleration is essential for many astrophysical problems, e.g., CMB foreground, ionization of molecular clouds and all high energy phenomena. Recent advances in MHD turbulence call for revisions in the paradigm of cosmic ray transport. We use the models of magnetohydrodynamic turbulence that were tested in numerical simulation, in which turbulence is injected at large scale and cascades to to small scales. We shall address the issue of the transport of CRs, both parallel and perpendicular to the magnetic field and show that the issue of cosmic ray subdiffusion is only important for restricted cases when the ambient turbulence is far from that suggested by numerical simulations. Moreover, on scales less than injection scale of turbulence, CRs's transport becomes super-diffusive. We also shall discuss the nonlinear growth of kinetic gyroresonance instability of cosmic rays induced by large scale compressible turbulence. This gyroresonance of cosmic rays on turbulence is demonstrated an important scattering mechanism in addition to direct interaction with the compressible turbulence. The feedback of the instability on large scale turbulence cannot be neglected, and should be included in future simulations.Comment: 7 pages, 1 figure, submitted to ASTRONUM2011 conference proceedings, typos correcte

Superdiffusion of Cosmic Rays: Implications for Cosmic Ray Acceleration

Diffusion of cosmic rays (CRs) is the key process of understanding their propagation and acceleration. We employ the description of spatial separation of magnetic field lines in MHD turbulence in Lazarian & Vishniac (1999) to quantify the divergence of magnetic field on scales less than the injection scale of turbulence and show this divergence induces superdiffusion of CR in the direction perpendicular to the mean magnetic field. The perpendicular displacement squared increases, not as distance $x$ along magnetic field, which is the case for a regular diffusion, but as the $x^{3}$ for freely streaming CRs. The dependence changes to $x^{3/2}$ for the CRs propagating diffusively along magnetic field. In the latter case we show that it is important to distinguish the perpendicular displacement in respect to the mean field and to the local magnetic field. We consider how superdiffusion changes the acceleration of CRs in shocks and show how it decreases efficiency of the CRs acceleration in perpendicular shocks. We also demonstrate that in the case when small-scale magnetic field is being generated in the pre-shock region, an efficient acceleration can take place for the CRs streaming without collisions along magnetic loops.Comment: 18 pages, 6 figures, erratum in eqs.(12,13) and table 1 included, ApJ accepte

The Effects of Eicosapentaenoic Acid (EPA) and Docosahexaenoic Acid (DHA) on Brown Adipogenesis in Stem Cell Culture

Polyunsaturated fatty acids (PUFAs), especially eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA), are major maternal dietary supplements due to their positive benefits on neurological tissue growth during the first 12 weeks of gestation. Previous studies show that EPA and DHA inhibit muscle formation but promote adipogenesis. However, no research has addressed the question whether high intake of EPA and DHA affects brown fat development during gestation. The objective of this study was to measure the effect of EPA and DHA supplement on brown adipogenesis and potential pathways related to mitochondrial biosynthesis using fibroblasts as in vitro model. Using Oil-Red-O staining and PCR testing, lipid droplet formation and tested six genes were examined and PGC1α presented statistically significant difference from the control group when treated with PUFAs. Results indicated that PGC1α gene expression can be to be alternated by EPA and DHA treatment. Mitochondrial biosynthesis can potentially be promoted by increased PGC1α gene expression. However, the lipid droplets accumulated in the PUFAs treated group show an unknown mechanism of the n-3 PUFA on adipogenesis that needs to be revealed